Prosecution Insights
Last updated: April 19, 2026
Application No. 17/652,464

PROBIOTIC THERAPIES FOR SOCIAL DEFICIT AND STRESS RESPONSE

Non-Final OA §103
Filed
Feb 24, 2022
Examiner
CRUM, MARY ABOU NADER
Art Unit
1657
Tech Center
1600 — Biotechnology & Organic Chemistry
Assignee
California Institute Of Technology
OA Round
7 (Non-Final)
41%
Grant Probability
Moderate
7-8
OA Rounds
3y 6m
To Grant
99%
With Interview

Examiner Intelligence

Grants 41% of resolved cases
41%
Career Allow Rate
32 granted / 78 resolved
-19.0% vs TC avg
Strong +68% interview lift
Without
With
+68.3%
Interview Lift
resolved cases with interview
Typical timeline
3y 6m
Avg Prosecution
42 currently pending
Career history
120
Total Applications
across all art units

Statute-Specific Performance

§101
7.7%
-32.3% vs TC avg
§103
38.6%
-1.4% vs TC avg
§102
10.8%
-29.2% vs TC avg
§112
23.7%
-16.3% vs TC avg
Black line = Tech Center average estimate • Based on career data from 78 resolved cases

Office Action

§103
Notice of Pre-AIA or AIA Status The present application, filed on or after March 16, 2013, is being examined under the first inventor to file provisions of the AIA . DETAILED ACTION Claims 1-3, 5-12, 26-27 are pending. Continued Examination Under 37 CFR 1.114 A request for continued examination under 37 CFR 1.114, including the fee set forth in 37 CFR 1.17(e), was filed in this application after final rejection. Since this application is eligible for continued examination under 37 CFR 1.114, and the fee set forth in 37 CFR 1.17(e) has been timely paid, the finality of the previous Office action has been withdrawn pursuant to 37 CFR 1.114. Applicant's submission filed on 11/03/2025 has been entered. Specification The disclosure is objected to because of the following informalities: Paragraph [0003] of the specification states “The Sequence Listing is provided as file CALTE158_SEQLIST.TXT created and last modified on February 17, 2022, which is 1,090 bytes in size”. However, the file name is 17652464_1_1.txt, with receipt date of 2/24/2022 and a size of 1107 bytes. Appropriate correction is required. Claim Objections Claims 1 and 27 are objected to because of the following informalities: The gene symbols “Crh1, c-Fos, and Nr3c1” should be replaced by the full name the first time they appear. Furthermore, gene symbols should be italicized. Appropriate correction is required. Claim Rejections - 35 USC § 103 The following is a quotation of 35 U.S.C. 103 which forms the basis for all obviousness rejections set forth in this Office action: A patent for a claimed invention may not be obtained, notwithstanding that the claimed invention is not identically disclosed as set forth in section 102, if the differences between the claimed invention and the prior art are such that the claimed invention as a whole would have been obvious before the effective filing date of the claimed invention to a person having ordinary skill in the art to which the claimed invention pertains. Patentability shall not be negated by the manner in which the invention was made. The factual inquiries for establishing a background for determining obviousness under 35 U.S.C. 103 are summarized as follows: 1. Determining the scope and contents of the prior art. 2. Ascertaining the differences between the prior art and the claims at issue. 3. Resolving the level of ordinary skill in the pertinent art. 4. Considering objective evidence present in the application indicating obviousness or nonobviousness. Claims 1-3, 5-12 and 26 are rejected under 35 U.S.C. 103 as being unpatentable over Arias (Psychoneuroendocrinology 42 (2014): 207-217, of record in Office Correspondence mailed on 09/20/2024) in view of Cambejun (WO2021010118, published 01-2021, of record in IDS filed on 04/13/2023), Lenze (The American Journal of Geriatric Psychiatry 19.5 (2011): 482-490, of record in Office Correspondence mailed on 03/04/2025), and Makino (Brain research 1048.1-2 (2005): 131-137) and as evidenced by Raff (Endocrinology 157.9 (2016): 3307-3308, of record in Office Correspondence mailed on 03/04/2025). Regarding claim 1, Arias teaches stress-sensitive Germfree (GF) rats have higher serum corticosterone concentrations in response to the open-field stress (i.e. identifying a subject in need of improving one or more symptoms of behavioral deficit) (Abstract, Fig. 3). Arias teaches the GF rats have increased corticosterone levels compared to specific pathogen free (SPF) rats (i.e. detecting a level of corticosterone in the subject, wherein the subject has increased corticosterone levels compared to a healthy subject) (Fig. 3). Arias teaches consumption of the Lactobacillus rhamnosus JB-1 probiotic strain lowered the stress-induced corticosterone secretion and toned down the level of anxiety-like behavior in the elevated plus maze test (i.e. detecting the level of corticosterone in the subject after administration of the composition, wherein the subject has decreased corticosterone levels compared to the corticosterone levels before administration of the composition) (page 214 right column para. 1). Arias does not teach administering an effective amount of one or more Enterococcus bacteria. However, Cambejun teaches germ-free mice exhibit enhanced corticosterone and adrenocorticotropic hormone in response to restraint stress than normal SPF mice ([0003]). Cambejun teaches administering a composition comprising bacteria belonging to the genus Enterococcus as an active ingredient and does not teach the presence of other bacteria (i.e., wherein the effective amount of the one or more Enterococcus bacteria is substantially free of bacteria other than the Enterococcus bacteria), and teaches the composition improves anxiety disorders and/or mood disorders ([0001]). Cambejun teaches the dose of the bacterium Enterococcus is 0.5x109 /kg body weight ([0045]). Applicant discloses an effective amount of Enterococcus bacteria is at least about 107 CFU (See specification [0012] and [0102]). Thus, the amount of composition taught by Cambejun falls within the limitation of “effective amount”. The limitations “thereby reducing corticosterone levels in the subject” and “wherein the one or more symptoms of behavioral deficit of the subject in need is improved after administering the one or more Enterococcus bacteria” do not require steps to be performed, and therefore, do not limit the method claimed. The whereby clause in a method claim is not given weight when it simply expresses the intended result of a process step positively recited. Both Arias and Cambejun teach subject with anxiety have increased corticosterone levels. Thus, the subject population taught by Arias and Cambejun is the same subject population instantly claimed. In addition, Lenze teaches treatment of anxiety disorder reduces the elevated cortisol levels in adults suffering from the disorder (title). Evidentiary reference Raff reports cortisol is the primary endogenous adrenal steroid in most mammals, including humans, whereas corticosterone is the primary adrenal corticosteroid in laboratory rodents (page 3307 left column second para.). Since Cambejun teaches the administration of the probiotic treated anxiety in the subject, one of ordinary skill in the art would expect a reduction of corticosterone levels in view of the teachings of Lenze and Arias. It would have been prima facie obvious to one of ordinary skill in the art before the effective filing date of the claimed invention to modify the method taught by Arias by administering an effective amount of a composition comprising bacteria belonging to the genus Enterococcus as an active ingredient to patients with anxiety as taught by Cambejun. One of ordinary skill in the art would be motivated to do so in order to improve the anxiety in the subject as taught by Cambejun. A skilled artisan would have a reasonable expectation of success in combining the teachings of these references to achieve the predictable outcome of improving one or more symptoms of behavioral deficit in a subject in need thereof. Arias does not teach detecting a level of a product of Crh1 gene. However, Makino teaches detecting the expression level of type 1 corticotropin-releasing hormone receptor mRNA which is increased during stress (Abstract). Applicant discloses Crh1 is Corticotropin-Releasing Hormone Receptor 1 gene (Remarks 11/03/2025 page 6). It would have been prima facie obvious to one of ordinary skill in the art before the effective filing date of the claimed invention to further modify the method taught by Arias by detecting the mRNA levels of type 1 corticotropin-releasing hormone receptor, as suggested by Makino. One of ordinary skill in the art would be motivated to do so in order to detect biomarkers of stress and monitor the progress and effectiveness of the treatment. Since Arias teaches a desire to detect biomarkers of stress (corticosterone, CRF mRNA levels) and since Makino teaches type 1 corticotropin-releasing hormone receptor mRNA is a reliable stress indicator, there is a reasonable expectation of success. MPEP §2144.06(I) states that “[i]t is prima facie obvious to combine equivalents each of which is taught by the prior art to be useful for the same purpose. Regarding claim 2, Arias teaches the behavioral deficit is anxiety (Abstract). Regarding claim 3, Cambejun does not specifically teach Enterococcus is the sole active ingredient. However, one of ordinary skill in the art can be motivated to administer the bacterium Enterococcus faecalis as the sole active ingredient since Cambejun teaches this bacterium is an active ingredient (claim 4), thus suggesting no other active ingredients are needed for the composition to be effective. Regarding claim 5, Cambejun teaches the composition comprises E. faecalis (claim 4). Regarding claim 6, Cambejun teaches the active ingredient is E. faecalis (claim 4). Regarding claim 7, Cambejun does not explicitly teach the composition is substantially free of bacteria other than the Enterococcus bacteria. However, Cambejun does not teach other bacteria are in the composition either. Cambejun teaches Enterococcus is an active ingredient and teaches the mice where fed AIN93-M (rodent feed) containing Enterococcus faecalis EC-12 strain ([0050]). One of ordinary skill in the art would be motivated to use a composition substantially free of bacteria other than Enterococcus faecalis since Cambejun does not teach other bacteria are needed for the composition to be effective. In addition, the addition of other bacteria might negatively affect the therapeutic effect of Enterococcus faecalis, thus rendering the composition less effective. Regarding claim 8, Cambejun teaches the composition can be taken orally ([0038]). Regarding claim 9, Arias teaches behavioral testing such as open field testing, sniffing, crawling, and following (Fig.1 and 2) Regarding claim 10, Cambejun teaches the composition is a functional food classified as probiotic (paragraph [0041]). Regarding claim 11, Cambejun teaches the composition is a health food ([0041]). Regarding claim 12, Cambejun teaches the composition is used as a pharmaceutical ([0022]). Regarding claim 26, Cambejun teaches the dose of the bacterium Enterococcus is 0.5x109 /kg body weight (i.e., at least about 107 colony forming units) ([0045]). Claim 27 is rejected under 35 U.S.C. 103 as being unpatentable over Arias (Psychoneuroendocrinology 42 (2014): 207-217, of record in Office Correspondence mailed on 09/20/2024) in view of Cambejun (WO2021010118, published 01-2021, of record in IDS filed on 04/13/2023), Lenze (The American Journal of Geriatric Psychiatry 19.5 (2011): 482-490, of record in Office Correspondence mailed on 03/04/2025), Makino (Brain research 1048.1-2 (2005): 131-137), and Zimprich (Frontiers in behavioral neuroscience 8 (2014): 125, of record in IDS filed on 04/13/2023) and as evidenced by Raff (Endocrinology 157.9 (2016): 3307-3308, of record in Office Correspondence mailed on 03/04/2025). The teachings of Arias, Cambejun, Lenze and Makino are discussed above and applied herein. Arias does not teach administering to the subject in need a corticosterone synthesis inhibitor, thereby reducing corticosterone levels in the subject. However, Zimprich teaches identifying stressed mice with symptoms of behavioral deficit (page 3 para. “stress protocol”, Figure 2), detecting the level of corticosterone (Figure 4) and administering a corticosterone synthesis inhibitor metyrapone to the mice under acute stress (Figure 5). Zimprich teaches the stress-induced hyperlocomotion is blocked by metyrapone (Abstract, Figure 5, page 125 left column first para.). It would have been prima facie obvious to one of ordinary skill in the art before the effective filing date of the claimed invention to further modify the method taught by Arias by administering to the subject metyrapone, a corticosterone synthesis inhibitor, as suggested by Zimprich. One of ordinary skill in the art would be motivated to do so in order to block the stress-induced behavior. Since Arias and Zimprich teach a desire to treat behavioral deficit related to high level of corticosterone, there is a reasonable expectation of success. The limitation of “thereby reducing corticosterone levels in the subject” expresses the result of the active step positively recited and does not limit the claim. Response to Arguments Applicant's arguments filed 11/03/2025 have been fully considered but they are not persuasive. Applicant argues Arias teaches detecting CRF mRNA but not Crh1 mRNA. In response to the argument, newly cited reference Makino teaches detecting the expression level of type 1 corticotropin-releasing hormone receptor mRNA which is increased during stress (Abstract). Applicant discloses Crh1 is Corticotropin-Releasing Hormone Receptor 1 gene (Remarks 11/03/2025 page 6). Since Arias teaches a desire to detect biomarkers of stress (corticosterone, CRF mRNA levels) and since Makino teaches type 1 corticotropin-releasing hormone receptor mRNA is a reliable stress indicator, there is a reasonable expectation of success. Applicant argues Arias, Cambejun, Lenze, and Zimprich teaches detection of stress via different methods and making their teachings not directly interchangeable, and argues a POSITA would not have a reasonable expectation of success in combination the teachings of the references and detecting gene products from Crh1, c-Fos, and Nr3c1 in the context of improving behavioral deficits as claimed. In response to the argument, Arias, Cambejun, Lenze, and Zimprich teach several markers for stress. MPEP §2144.06(I) states it is obvious to combine equivalents know for the same purpose. Since these markers are known as indicators of stress, then there is a reasonable expectation of success. Cambejun teaches the claimed step of administering to the subject in need, an effective amount of Enterococcus bacteria and teaches detecting the expression of neurotransmitter receptor genes in the subject. Thus, one skilled in the art would be motivated, and have a reasonable expectation of success, to administer the composition to treat the anxiety disorder and to detect biomarkers in order to monitor the progress and effectiveness of the treatment. Conclusion Any inquiry concerning this communication or earlier communications from the examiner should be directed to MARY A CRUM whose telephone number is (571)272-1661. The examiner can normally be reached M-F 8:00-5:00 CT with alternate Fridays off. Examiner interviews are available via telephone, in-person, and video conferencing using a USPTO supplied web-based collaboration tool. To schedule an interview, applicant is encouraged to use the USPTO Automated Interview Request (AIR) at http://www.uspto.gov/interviewpractice. If attempts to reach the examiner by telephone are unsuccessful, the examiner’s supervisor, LOUISE W HUMPHREY can be reached at 571-272-5543. The fax phone number for the organization where this application or proceeding is assigned is 571-273-8300. Information regarding the status of published or unpublished applications may be obtained from Patent Center. Unpublished application information in Patent Center is available to registered users. To file and manage patent submissions in Patent Center, visit: https://patentcenter.uspto.gov. Visit https://www.uspto.gov/patents/apply/patent-center for more information about Patent Center and https://www.uspto.gov/patents/docx for information about filing in DOCX format. For additional questions, contact the Electronic Business Center (EBC) at 866-217-9197 (toll-free). If you would like assistance from a USPTO Customer Service Representative, call 800-786-9199 (IN USA OR CANADA) or 571-272-1000. /MARY A CRUM/Examiner, Art Unit 1657 /THANE UNDERDAHL/Primary Examiner, Art Unit 1699
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Prosecution Timeline

Feb 24, 2022
Application Filed
May 03, 2023
Non-Final Rejection — §103
Aug 08, 2023
Response Filed
Aug 21, 2023
Final Rejection — §103
Jan 11, 2024
Request for Continued Examination
Jan 17, 2024
Response after Non-Final Action
Apr 22, 2024
Non-Final Rejection — §103
Jul 25, 2024
Response Filed
Sep 19, 2024
Final Rejection — §103
Dec 17, 2024
Request for Continued Examination
Dec 20, 2024
Response after Non-Final Action
Feb 26, 2025
Non-Final Rejection — §103
Jun 18, 2025
Interview Requested
Jun 26, 2025
Examiner Interview Summary
Jul 03, 2025
Response Filed
Jul 29, 2025
Final Rejection — §103
Nov 03, 2025
Request for Continued Examination
Nov 04, 2025
Response after Non-Final Action
Dec 18, 2025
Non-Final Rejection — §103 (current)

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Study what changed to get past this examiner. Based on 5 most recent grants.

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Prosecution Projections

7-8
Expected OA Rounds
41%
Grant Probability
99%
With Interview (+68.3%)
3y 6m
Median Time to Grant
High
PTA Risk
Based on 78 resolved cases by this examiner. Grant probability derived from career allow rate.

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